Wilson’s Temperature Syndrome may be a fresh idea to some, but the tenets of this protocol are well-established and backed by decades of clinical studies and research. The connection between low body temperature, low thyroid function, and a host of associated far-reaching symptoms have been on scientists’ radars for some time.
Consider some of the early animal studies related to low body temperature and low thyroid function. Researchers suggested very early on, way back in the 1970s, that genetically obese mice can be identified by their low oxygen consumption and low body temperature, already hinting at the metabolism connection. Around the same time, researchers suggested that mice with low thyroid function seemed to have malfunctioning enzymes in their bodies — once again, echoing the tenets of WTS protocols.
In one early study conducted in the Netherlands, researchers investigated how well the thyroid functioned in obese mice with high blood sugar (obese-hyperglycemic syndrome). Researchers also investigated normal-weight mice as a control. Thyroid cell activity, iodine levels and T4 levels in blood were tested. After taking measurements, researchers concluded that the obese-hyperglycemic mouse was also hypothyroid. Researchers reached this conclusion because the mouse’s low body temperature went up to normal with the administration of thyroid hormone. They further suggested that the mouse’s hypothyroidism was one of the causes behind its obese-hyperglycemic syndrome.
It is intriguing to see how early animal research seemed to be juggling these puzzle pieces: Obesity, blood sugar issues, metabolic problems, enzyme malfunction, low body temperature and low thyroid function. Twenty years later, Wilson’s Temperature Syndrome presented a unifying theory that seems to fit all these pieces together and solve the puzzle.
Want to take action on your low body temp? See a WTS doctor for hormone treatments to address all the issues above.
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